The erosive effect of various drinks, foods, stimulants, medications and mouthwashes on human tooth enamel.

Two forms of non-carious dental disorder - erosive tooth hard tissue loss and dental erosion - have been increasingly observed in recent years. Dental erosion is the chemical loss of dental hard substances caused by exposure to acids not derived from oral bacteria. Mechanical forces from, for example, the tongue, the cheeks or toothbrushing, increase loss of partly-demineralized tooth surfaces and the cumulative loss of dental hard tissue is defined as erosive tooth wear (ETW). Dental hard tissue losses which occur because of very frequent acid exposure, such as through increased vomiting, but without mechanical stress, are also assigned to tooth erosion. Without prior softening, practically no loss of enamel takes place due to abrasion with the modern Western diet. The present work is a continuation of earlier work. A total of 226 beverages, food, stimulants as well as medicines and mouthwashes were tested for their erosive potential on premolars and deciduous molars covered with a human pellicle. The influence of temperature, phosphate and calcium was also investigated in additional experiments. The change in hardness before and after immersion in the respective test substance was measured and the erosive potential was classified. For each test product, we determined pH and other properties which were possibly related to erosive potential. There were considerable and sometimes surprising differences between the tested products. The addition of phosphate did not influence the erosive potential of the liquids, but calcium did. A modified erosion scheme is presented, which incorporates these and other new findings.

The erosive potential of different drinks, foodstuffs and medicines ­ a vade mecum / Das erosive Potenzial verschiedener Getränke, Speisen und Medikamente. Ein Vademecum

There is increasing evidence that the excessive consumption of acidic drinks and foods contributes to dental erosion and may be an important contributing factor for erosive tooth wear. The aims of the present contribution were twofold: (1) to assess the erosive potential of 116 dietary substances and medications; (2) to determine the chemical properties with an impact on the erosive potential. Using 300 deciduous and 1,020 permanent human teeth, enamel specimens were prepared and a pellicle was formed with human saliva. The erosive potential of the tested agents was quantified as the change in surface hardness of the specimens after 2 min of erosion. To characterise these agents, the following chemical properties were determined: pH, titratable acidity to pH 7, concentrations of Ca, Pi and F, as well as the degree of saturation with respect to hydroxyapatite. We conclude that some drinks, foodstuffs and medications may cause erosion. However, pH is not the only decisive factor, since some acidic substances did not cause dental erosion.

Effects of enamel abrasion, salivary pellicle, and measurement angle on the optical assessment of dental erosion.

The present study assessed the effects of abrasion, salivary proteins, and measurement angle on the quantification of early dental erosion by the analysis of reflection intensities from enamel. Enamel from 184 caries-free human molars was used for in vitro erosion in citric acid (pH 3.6). Abrasion of the eroded enamel resulted in a 6% to 14% increase in the specular reflection intensity compared to only eroded enamel, and the reflection increase depended on the erosion degree. Nevertheless, monitoring of early erosion by reflection analysis was possible even in the abraded eroded teeth. The presence of the salivary pellicle induced up to 22% higher reflection intensities due to the smoothing of the eroded enamel by the adhered proteins. However, this measurement artifact could be significantly minimized (p<0.05) by removing the pellicle layer with 3% NaOCl solution. Change of the measurement angles from 45 to 60 deg did not improve the sensitivity of the analysis at late erosion stages. The applicability of the method for monitoring the remineralization of eroded enamel remained unclear in a demineralization/remineralization cycling model of early dental erosion in vitro.

Analysis of the erosive effect of different dietary substances and medications.

Excessive consumption of acidic drinks and foods contributes to tooth erosion. The aims of the present in vitro study were twofold: (1) to assess the erosive potential of different dietary substances and medications; (2) to determine the chemical properties with an impact on the erosive potential. We selected sixty agents: soft drinks, an energy drink, sports drinks, alcoholic drinks, juice, fruit, mineral water, yogurt, tea, coffee, salad dressing and medications. The erosive potential of the tested agents was quantified as the changes in surface hardness (ΔSH) of enamel specimens within the first 2 min (ΔSH2-0 = SH2 min - SHbaseline) and the second 2 min exposure (ΔSH4-2 = SH4 min - SH2 min). To characterise these agents, various chemical properties, e.g. pH, concentrations of Ca, Pi and F, titratable acidity to pH 7·0 and buffering capacity at the original pH value (ß), as well as degree of saturation (pK - pI) with respect to hydroxyapatite (HAP) and fluorapatite (FAP), were determined. Erosive challenge caused a statistically significant reduction in SH for all agents except for coffee, some medications and alcoholic drinks, and non-flavoured mineral waters, teas and yogurts (P < 0·01). By multiple linear regression analysis, 52 % of the variation in ΔSH after 2 min and 61 % after 4 min immersion were explained by pH, ß and concentrations of F and Ca (P < 0·05). pH was the variable with the highest impact in multiple regression and bivariate correlation analyses. Furthermore, a high bivariate correlation was also obtained between (pK - pI)HAP, (pK - pI)FAP and ΔSH.

The effect of magnification on the iatrogenic damage to adjacent tooth surfaces during class II preparation.

OBJECTIVES: To determine the amount of damage to adjacent surfaces during cavity preparation of approximal box-cavities by using either no magnification or an individually adapted surgical telescope. MATERIALS AND METHODS: Nine dentists prepared in a mannequin head 4 approximal (class II) box-cavities without magnification using a high speed handpiece with lighting from a dental unit. At least 2 months later the test was repeated with a surgical telescope system with integrated light. The degree of damage the 72 adjacent surfaces suffered was determined. RESULTS: The average time needed to prepare one cavity was 14.9min without and 18.3min with magnification (p=0.01). Altogether, 29.7% of adjacent area were damaged when no surgical telescopes were used and 34.5% when surgical telescopes were applied (p>0.05). When the mesial-facing and distal-facing surfaces were analysed independently a statistically significant increase of damage was found on distal surfaces (p=0.03) when using surgical telescopes. Seventy surfaces (=97%) had a preparation trauma. CONCLUSIONS: Surgical telescopes do not decrease damage of adjacent tooth surfaces.